Amyloid Aβ(25-35) Aggregates Say ‘NO’ to Long-Term Potentiation in the Hippocampus through Activation of Stress-Induced Phosphatase 1 and Mitochondrial Na(+)/Ca(2+) Exchanger

The search for strategies for strengthening the synaptic efficiency in Aβ(25-35)-treated slices is a challenge for the compensation of amyloidosis-related pathologies. Here, we used the recording of field excitatory postsynaptic potentials (fEPSPs), nitric oxide (NO) imaging, measurements of serine/...

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Detalles Bibliográficos
Autores principales: Maltsev, Alexander V., Nikiforova, Anna B., Bal, Natalia V., Balaban, Pavel M.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: MDPI 2022
Materias:
Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9570122/
https://www.ncbi.nlm.nih.gov/pubmed/36233148
http://dx.doi.org/10.3390/ijms231911848

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9570122/
https://www.ncbi.nlm.nih.gov/pubmed/36233148
http://dx.doi.org/10.3390/ijms231911848

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