Inhibiting amyloid beta (1–42) peptide-induced mitochondrial dysfunction prevents the degradation of synaptic proteins in the entorhinal cortex

Increasing evidence suggests that mitochondrial dysfunction and aberrant release of mitochondrial reactive oxygen species (ROS) play crucial roles in early synaptic perturbations and neuropathology that drive memory deficits in Alzheimer’s disease (AD). We recently showed that solubilized human amyl...

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Detalles Bibliográficos
Autores principales: Olajide, Olayemi Joseph, La Rue, Claudia, Bergdahl, Andreas, Chapman, Clifton Andrew
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2022
Materias:
Aging Neuroscience
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9582742/
https://www.ncbi.nlm.nih.gov/pubmed/36275011
http://dx.doi.org/10.3389/fnagi.2022.960314

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9582742/
https://www.ncbi.nlm.nih.gov/pubmed/36275011
http://dx.doi.org/10.3389/fnagi.2022.960314

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