Cargando…
Crosstalk between age accumulated DNA-damage and the SIRT1-AKT-GSK3ß axis in urine derived renal progenitor cells
The aging process is manifested by a multitude of inter-linked biological processes. These processes contribute to genomic instability, telomere attrition, epigenetic alterations, loss of proteostasis, de-regulated nutrient-sensing, mitochondrial dysfunction, cellular senescence, stem cell exhaustio...
Autores principales: | Erichsen, Lars, Adjaye, James |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Impact Journals
2022
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9648809/ https://www.ncbi.nlm.nih.gov/pubmed/36170022 http://dx.doi.org/10.18632/aging.204300 |
Ejemplares similares
-
The FGF, TGFβ and WNT axis Modulate Self-renewal of Human SIX2(+) Urine Derived Renal Progenitor Cells
por: Rahman, Md Shaifur, et al.
Publicado: (2020) -
Secreted Cytokines within the Urine of AKI Patients Modulate TP53 and SIRT1 Levels in a Human Podocyte Cell Model
por: Erichsen, Lars, et al.
Publicado: (2023) -
Unveiling Angiotensin II and Losartan-Induced Gene Regulatory Networks Using Human Urine-Derived Podocytes
por: Thimm, Chantelle, et al.
Publicado: (2023) -
Activation of the Renin–Angiotensin System Disrupts the Cytoskeletal Architecture of Human Urine-Derived Podocytes
por: Erichsen, Lars, et al.
Publicado: (2022) -
JNK Signalling Regulates Self-Renewal of Proliferative Urine-Derived Renal Progenitor Cells via Inhibition of Ferroptosis
por: Nguyen, Lisa, et al.
Publicado: (2023)