Methyltransferase like 3-mediated N6-methylatidin methylation inhibits vascular smooth muscle cells phenotype switching via promoting phosphatidylinositol 3-kinase mRNA decay

N6-methylatidine (m6A) is involved in post-transcriptional metabolism and a variety of pathological processes. However, little is known about the role of m6A in vascular proliferative diseases, particularly in vascular smooth muscle cells (VSMCs) phenotype switching-induced neointimal hyperplasia. I...

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Detalles Bibliográficos
Autores principales: Zhao, Yongchao, Xia, Aichao, Li, Chaofu, Long, Xianping, Bai, Zhixun, Qiu, Zhimei, Xiong, Weidong, Gu, Ning, Shen, Youcheng, Zhao, Ranzun, Shi, Bei
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2022
Materias:
Cardiovascular Medicine
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9649646/
https://www.ncbi.nlm.nih.gov/pubmed/36386358
http://dx.doi.org/10.3389/fcvm.2022.913039

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9649646/
https://www.ncbi.nlm.nih.gov/pubmed/36386358
http://dx.doi.org/10.3389/fcvm.2022.913039

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