IL‐17A promotes lung fibrosis through impairing mitochondrial homeostasis in type II alveolar epithelial cells

The dysfunction of type II alveolar epithelial cells (AECIIs), mainly manifested by apoptosis, has emerged as a major component of idiopathic pulmonary fibrosis (IPF) pathophysiology. A pivotal mechanism leading to AECIIs apoptosis is mitochondrial dysfunction. Recently, interleukin (IL)‐17A has bee...

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Detalles Bibliográficos
Autores principales: Xiao, Huijuan, Peng, Liang, Jiang, Dingyuan, Liu, Yuan, Zhu, Lili, Li, Zhen, Geng, Jing, Xie, Bingbing, Huang, Xiaoxi, Wang, Jing, Dai, Huaping, Wang, Chen
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2022
Materias:
Original Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9667517/
https://www.ncbi.nlm.nih.gov/pubmed/36308405
http://dx.doi.org/10.1111/jcmm.17600

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9667517/
https://www.ncbi.nlm.nih.gov/pubmed/36308405
http://dx.doi.org/10.1111/jcmm.17600

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