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IL‐17A promotes lung fibrosis through impairing mitochondrial homeostasis in type II alveolar epithelial cells
The dysfunction of type II alveolar epithelial cells (AECIIs), mainly manifested by apoptosis, has emerged as a major component of idiopathic pulmonary fibrosis (IPF) pathophysiology. A pivotal mechanism leading to AECIIs apoptosis is mitochondrial dysfunction. Recently, interleukin (IL)‐17A has bee...
Autores principales: | Xiao, Huijuan, Peng, Liang, Jiang, Dingyuan, Liu, Yuan, Zhu, Lili, Li, Zhen, Geng, Jing, Xie, Bingbing, Huang, Xiaoxi, Wang, Jing, Dai, Huaping, Wang, Chen |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
John Wiley and Sons Inc.
2022
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9667517/ https://www.ncbi.nlm.nih.gov/pubmed/36308405 http://dx.doi.org/10.1111/jcmm.17600 |
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