Chronic exposure to carbon black ultrafine particles reprograms macrophage metabolism and accelerates lung cancer

Chronic exposure to airborne carbon black ultrafine (nCB) particles generated from incomplete combustion of organic matter drives IL-17A–dependent emphysema. However, whether and how they alter the immune responses to lung cancer remains unknown. Here, we show that exposure to nCB particles increase...

Descripción completa

Detalles Bibliográficos
Autores principales: Chang, Cheng-Yen, You, Ran, Armstrong, Dominique, Bandi, Ashwini, Cheng, Yi-Ting, Burkhardt, Philip M., Becerra-Dominguez, Luis, Madison, Matthew C., Tung, Hui-Ying, Zeng, Zhimin, Wu, Yifan, Song, Lizhen, Phillips, Patricia E., Porter, Paul, Knight, John M., Putluri, Nagireddy, Yuan, Xiaoyi, Marcano, Daniela C., McHugh, Emily A., Tour, James M., Catic, Andre, Maneix, Laure, Burt, Bryan M., Lee, Hyun-Sung, Corry, David B., Kheradmand, Farrah
Formato: Online Artículo Texto
Lenguaje:English
Publicado: American Association for the Advancement of Science 2022
Materias:
Biomedicine and Life Sciences
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9668323/
https://www.ncbi.nlm.nih.gov/pubmed/36383649
http://dx.doi.org/10.1126/sciadv.abq0615

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9668323/
https://www.ncbi.nlm.nih.gov/pubmed/36383649
http://dx.doi.org/10.1126/sciadv.abq0615

Ejemplares similares