Increased AR expression in castration-resistant prostate cancer rapidly induces AR signaling reprogramming with the collaboration of EZH2

Elevated androgen receptor (AR) expression is a hallmark of castration-resistant prostate cancer (CRPC) and contributes to the restoration of AR signaling under the conditions of androgen deprivation. However, whether overexpressed AR alone with the stimulation of castrate levels of androgens can be...

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Detalles Bibliográficos
Autores principales: Labaf, Maryam, Li, Muqing, Ting, Lily, Karno, Breelyn, Zhang, Songqi, Gao, Shuai, Patalano, Susan, Macoska, Jill A., Zarringhalam, Kourosh, Han, Dong, Cai, Changmeng
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Frontiers Media S.A. 2022
Materias:
Oncology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9669968/
https://www.ncbi.nlm.nih.gov/pubmed/36408179
http://dx.doi.org/10.3389/fonc.2022.1021845

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9669968/
https://www.ncbi.nlm.nih.gov/pubmed/36408179
http://dx.doi.org/10.3389/fonc.2022.1021845

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