Mettl3-mediated m(6)A modification of Fgf16 restricts cardiomyocyte proliferation during heart regeneration

Cardiovascular disease is the leading cause of death worldwide due to the inability of adult heart to regenerate after injury. N(6)-methyladenosine (m(6)A) methylation catalyzed by the enzyme methyltransferase-like 3 (Mettl3) plays an important role in various physiological and pathological bioproce...

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Detalles Bibliográficos
Autores principales: Jiang, Fu-Qing, Liu, Kun, Chen, Jia-Xuan, Cao, Yan, Chen, Wu-Yun, Zhao, Wan-Ling, Song, Guo-Hua, Liang, Chi-Qian, Zhou, Yi-Min, Huang, Huan-Lei, Huang, Rui-Jin, Zhao, Hui, Park, Kyu-Sang, Ju, Zhenyu, Cai, Dongqing, Qi, Xu-Feng
Formato: Online Artículo Texto
Lenguaje:English
Publicado: eLife Sciences Publications, Ltd 2022
Materias:
Cell Biology
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9674341/
https://www.ncbi.nlm.nih.gov/pubmed/36399125
http://dx.doi.org/10.7554/eLife.77014

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9674341/
https://www.ncbi.nlm.nih.gov/pubmed/36399125
http://dx.doi.org/10.7554/eLife.77014

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