Activin A and CCR2 regulate macrophage function in testicular fibrosis caused by experimental autoimmune orchitis

Experimental autoimmune-orchitis (EAO), a rodent model of chronic testicular inflammation and fibrosis, replicates pathogenic changes seen in some cases of human spermatogenic disturbances. During EAO, increased levels of pro-inflammatory and pro-fibrotic mediators such as TNF, CCL2, and activin A a...

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Detalles Bibliográficos
Autores principales: Peng, Wei, Kepsch, Artem, Kracht, Till O., Hasan, Hiba, Wijayarathna, Rukmali, Wahle, Eva, Pleuger, Christiane, Bhushan, Sudhanshu, Günther, Stefan, Kauerhof, A. Christine, Planinić, Ana, Fietz, Daniela, Schuppe, Hans-Christian, Wygrecka, Małgorzata, Loveland, Kate L., Ježek, Davor, Meinhardt, Andreas, Hedger, Mark P., Fijak, Monika
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Springer International Publishing 2022
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9700630/
https://www.ncbi.nlm.nih.gov/pubmed/36434305
http://dx.doi.org/10.1007/s00018-022-04632-4

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9700630/
https://www.ncbi.nlm.nih.gov/pubmed/36434305
http://dx.doi.org/10.1007/s00018-022-04632-4

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