Selective autophagy of RIPosomes maintains innate immune homeostasis during bacterial infection

The NOD1/2‐RIPK2 is a key cytosolic signaling complex that activates NF‐κB pro‐inflammatory response against invading pathogens. However, uncontrolled NF‐κB signaling can cause tissue damage leading to chronic diseases. The mechanisms by which the NODs‐RIPK2‐NF‐κB innate immune axis is activated and...

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Detalles Bibliográficos
Autores principales: Mehto, Subhash, Jena, Kautilya Kumar, Yadav, Rina, Priyadarsini, Swatismita, Samal, Pallavi, Krishna, Sivaram, Dhar, Kollori, Jain, Ashish, Chauhan, Nishant Ranjan, Murmu, Krushna C, Bal, Ramyasingh, Sahu, Rinku, Jaiswal, Pundrik, Sahoo, Bhabani Sankar, Patnaik, Srinivas, Kufer, Thomas A, Rusten, Tor Erik, Chauhan, Swati, Prasad, Punit, Chauhan, Santosh
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2022
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9713718/
https://www.ncbi.nlm.nih.gov/pubmed/36221902
http://dx.doi.org/10.15252/embj.2022111289

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9713718/
https://www.ncbi.nlm.nih.gov/pubmed/36221902
http://dx.doi.org/10.15252/embj.2022111289

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