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Aβ plaques do not protect against HSV‐1 infection in a mouse model of familial Alzheimer's disease, and HSV‐1 does not induce Aβ pathology in a model of late onset Alzheimer's disease

The possibility that the etiology of late onset Alzheimer's disease is linked to viral infections of the CNS has been actively debated in recent years. According to the antiviral protection hypothesis, viral pathogens trigger aggregation of Aβ peptides that are produced as a defense mechanism i...

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Detalles Bibliográficos
Autores principales: Bocharova, Olga V., Fisher, Aidan, Pandit, Narayan P., Molesworth, Kara, Mychko, Olga, Scott, Alison J., Makarava, Natallia, Ritzel, Rodney, Baskakov, Ilia V.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: John Wiley and Sons Inc. 2022
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9836376/
https://www.ncbi.nlm.nih.gov/pubmed/36064300
http://dx.doi.org/10.1111/bpa.13116