Tranilast reduces cardiomyocyte injury induced by ischemia‑reperfusion via Nrf2/HO‑1/NF‑κB signaling

Tranilast, a synthetic derivative of a tryptophan metabolite, can be used to treat heart diseases. However, the specific mechanism underlying the effect of tranilast on ischemia-reperfusion (I/R) injury-induced cardiomyocyte apoptosis remains unclear. Therefore, the present study aimed to determine...

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Detalles Bibliográficos
Autores principales: Wang, Wei, Shen, Qifeng
Formato: Online Artículo Texto
Lenguaje:English
Publicado: D.A. Spandidos 2023
Materias:
Articles
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9996351/
https://www.ncbi.nlm.nih.gov/pubmed/36911371
http://dx.doi.org/10.3892/etm.2023.11859

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC9996351/
https://www.ncbi.nlm.nih.gov/pubmed/36911371
http://dx.doi.org/10.3892/etm.2023.11859

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